temperature delta low

Edge evidence

• limited thermal-adaptation flexibility produces temperature delta low METPO:2000202

  Limited thermal-adaptation flexibility yields a 5–10 °C temperature-delta breadth.

  • DOI:10.1146/annurev-micro-091313-103612 more unsaturated fatty acids Supports limited thermal-adaptation flexibility as the basis of narrow breadth.
• temperature delta low is a temperature delta rdfs:subClassOf

  Temperature delta low is a quantitative bin of the temperature-delta phenotype.

  • DOI:10.1016/s0300-9629(97)00003-0 adapted to environments of high temperature Supports the 5–10 °C breadth as a value within the temperature-delta distribution.
• temperature decrease causes membrane rigidification/thickening biolink:causes

  Cooling reduces bilayer fluidity, inducing membrane rigidification and thickening.

  • DOI:10.1128/spectrum.03925-23 Decreasing temperature induces membrane rigidification and thickening, producing a liquid-crystalline-to-gel phase transition.
• membrane unsaturated fatty acids contributes to homeoviscous adaptation / restored fluidity RO:0002326

  Increased membrane unsaturated fatty acids counter low-temperature rigidification and restore bilayer fluidity.

  • DOI:10.1146/annurev-micro-091313-103612 Bacteria perform homoviscous adaptation by increasing membrane unsaturated fatty acids as growth temperature decreases, thereby restoring bilayer fluidity.
• homeoviscous adaptation / restored fluidity counteracts membrane rigidification/thickening

  Homeoviscous adaptation restores fluidity, opposing cold-induced membrane rigidification.

  • DOI:10.1146/annurev-micro-091313-103612 Homeoviscous adaptation restores bilayer fluidity against rigidification as growth temperature decreases.
• temperature decrease shifts iso-to-anteiso branched-chain fatty acid shift

  Low-temperature acclimation drives iso-to-anteiso branched-chain fatty acid switching for membrane fluidization.

  • DOI:10.1007/s42770-023-01057-4 Predominant long-term adaptation to low temperatures occurs via fluidization of the membrane by chain branching, specifically iso-to-anteiso SFA switching.
• iso-to-anteiso branched-chain fatty acid shift contributes to homeoviscous adaptation / restored fluidity RO:0002326

  Branched-chain fatty acid remodeling fluidizes the membrane, supporting homeoviscous adaptation.

  • DOI:10.1007/s42770-023-01057-4 Long-term low-temperature adaptation fluidizes the membrane via introduction of chain branching, a homeoviscous mechanism.
• limited thermal-adaptation flexibility limits homeoviscous adaptation / restored fluidity RO:0002212

  Impaired acyl-chain remodeling capacity constrains homeoviscous adaptation flexibility, narrowing thermal breadth.

  • DOI:10.1101/2023.11.10.566608 Impaired homeoviscous adaptation: unable to regulate the acyl chain profile to the same extent in response to temperature fluctuations.