tailed shaped

Edge evidence

• unipolar peptidoglycan growth regulates stalk outgrowth RO:0002211

  Unipolar PG growth at the same cell pole drives stalk extension.

  • DOI:10.1146/annurev.micro.061705.103240 prosthecate bacteria Supports polar wall growth as the mechanism extending the stalk.
• stalk outgrowth produces polar appendage METPO:2000202

  Stalk outgrowth produces an elongated polar appendage.

  • DOI:10.1146/annurev.micro.061705.103240 prosthecate bacteria Supports stalk extension as a prosthecate-cell feature.
• polar appendage manifests as tailed shaped METPO:2007400

  A polar appendage manifests the tailed-shaped trait.

  • DOI:10.1146/annurev.micro.061705.103240 prosthecate bacteria Supports the trait endpoint.
• bactofilin polymers localizes to stalk base biolink:located_in

  Bactofilin polymers localize to the stalk base to direct proper stalk formation.

  • DOI:10.7554/eLife.86577 In C. crescentus and A. biprosthecum bactofilin polymers localize to the stalk base to direct proper stalk formation.
• bactofilin polymers regulates peptidoglycan biosynthesis at stalk base RO:0002211

  Bactofilins limit peptidoglycan biosynthesis to the stalk base, confining wall growth spatially.

  • DOI:10.7554/eLife.86577 Required to efficiently initiate stalk formation and limit peptidoglycan biosynthesis to the stalk base.
• bactofilin polymers recruits cell wall synthase

  Bactofilins recruit a cell wall synthase that contributes to stalk elongation.

  • DOI:10.7554/eLife.86577 In C. crescentus, they recruit a cell wall synthase that contributes to stalk elongation.
• cell wall synthase positively regulates stalk outgrowth

  The recruited cell wall synthase contributes to stalk elongation.

  • DOI:10.7554/eLife.86577 Recruited cell wall synthase contributes to stalk elongation.
• MreB required for stalk outgrowth

  MreB is necessary for stalk formation; depletion causes a stalk elongation defect.

  • DOI:10.1128/jb.00384-22 MreB and RodA are necessary for stalk formation, as depletion of either protein results in a stalk elongation defect.
• RodA required for stalk outgrowth

  RodA (SEDS PG polymerase) is necessary for stalk formation; depletion causes a stalk elongation defect.

  • DOI:10.1128/jb.00384-22 MreB and RodA are necessary for stalk formation, as depletion of either protein results in a stalk elongation defect.
• phosphate starvation induces stalk outgrowth

  Phosphate starvation induces elongated stalks.

  • DOI:10.1128/jb.00384-22 Cells starved of phosphate grow elongated stalks.